《2011ATA妊娠期甲狀腺疾病診療指南》內(nèi)容簡介：

    Pregnancy has a profound impact on the thyroid glandand thyroid function. The gland increases 10% in sizeduring pregnancy in iodine-replete countries and by 20%–40% in areas of iodine deficiency. Production of thyroxine（T4） and triiodothyronine （T3） increases by 50%, along with a50% increase in the daily iodine requirement.

    《2011ATA妊娠期甲狀腺疾病診療指南》內(nèi)容預(yù)覽：

    Knowledge regarding the interaction between the thyroidand pregnancy/the postpartum period is advancing at arapid pace. Only recently has a TSH of 2.5 mIU/L been ac-cepted as the upper limit of normal for TSH in the first tri-mester. This has important implications in regards tointerpretation of the literature as well as a critical impact forthe clinical diagnosis of hypothyroidism. Although it is wellaccepted that overt hypothyroidism and overt hyperthy-roidism have a deleterious impact on pregnancy, studies arenow focusing on the potential impact of subclinical hypo-thyroidism and subclinical hyperthyroidism on maternal andfetal health, the association between miscarriage and pretermdelivery in euthyroid women positive for TPO and/or Tgantibody, and the prevalence and long-term impact of post-partum thyroiditis. Recently completed prospective ran-domized studies have begun to produce critically needed dataon the impact of treating thyroid disease on the mother, fetus,and the future intellect of the unborn child.

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